Approach to C-spine x-rays
3-view C-spine series is the screening modality of choice:
1.
Lateral C1~T1 +/- swimmer’s view
a.
Lateral view is best; it identifies 90~95% of
injuries
2.
Odontoid view (open mouth or oblique submental
view)
a.
Examine the dens for fractures
i. If
unable to rule out fracture, repeat view or consider CT or plain film
tomography
b.
Examine lateral aspects of C1 and spacing
relative to C2
3.
AP view
a.
Alignment of spinous processes in the midline
b.
Spacing of spinous processes should be equal
c.
Check vertebral bodies and facet dislocations
Normal Swimmer’s view x-ray
The ABCS of lateral view
A—adequacy and alignment
·
Must see C1~T1
junction; if not, downward traction of shoulders, swimmer’s view, bilateral
supine oblique, or CT scan are needed.
·
Lines of contour
in children < 8 yr of age, can see physiologic subluxation of C2 on C3, and
C3 on C4, but the spino-laminal line is maintained.
·
Fanning of
spinous processes suggests posterior ligamentous disruption
·
Widening of facet
joints
·
Check
atlanto-occipital joint
·
Line extending
inferiorly from clivus should transect odontoid (Wackenheim or clivus-canal
line)
·
Atlanto-axial
articulation, widening of predental space (normal: < 3mm in adults; < 5mm
in children) indicates injury of C1 or C2
B—bones
·
Height, width,
and shape of each vertebral body
·
Pedicles, facets,
and laminae should appear as one—doubling suggests rotation
C—cartilage
·
Intervertebral
disc spaces—wedging anteriorly or posteriorly suggests vertebral compression
S—soft tissue
·
Widening of retropharyngeal space (normal: <
7 mm at C1~C4; may be wide in children < 2yrs. on expiration) or
retrotracheal spaces (normal: < 22mm at C6~T1; < 14mm in children <
5yrs.)
Supine oblique views, though rarely used, are better
visualization of posterior element fractures (lamina, pedicle, facet joint) and
is good for assessing the patency of neural foramina; it can also be used to
visualize the C7~T1 junction.
Sequelae of C-spine fractures:
·
Acute phase of spinal cord injury
o
Spinal shock—absence of all voluntary and reflex
activity below level of injury
§
Decreased reflexes, no sensation, flaccid
paralysis below level of injury, lasting days to months
o
Neurogenic shock—loss of vasomotor tone, SNS
tone
§
Watch for hypotension (lacking SNS), bradycardia
(unopposed PNS), and poikilothermia (inability to maintain constant core
temperature due to lacking SNS so no shunting of blood from extremities to
core).
§
Occurs within 30min of spinal cord injury at
level T6 or above, lasting up to 6 weeks
§
Provide airway support, fluids, atropine (for
bradycardia), vasopressors for BP support
·
Chronic phase
o
Autonomic dysreflexia—in patients with a spinal
cord injury at level T6 or above
§
Symptoms and signs include pounding headache,
nasal congestion, feeling of apprehension or anxiety, visual changes,
dangerously increased blood pressure. (Think opposite of neurogenic shock!)
§
Common triggers
·
GU causes—bladder distention, UTI, kidney stones
·
GI causes—fecal impaction or bowel distention
§
Treatment—monitoring and controlling blood pressure,
prior to addressing causative issue
20% of C-spine fractures are accompanied by other spinal
fractures so ensure thoracic and lumbar spinal x-rays are normal before
proceeding to operating theatre.
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