Running diagnostics

Running diagnostics

Friday, June 16, 2017

Eye Emergencies

 I have always felt that the eye has not been given the amount of time and detailed education that it deserves during med school training; and as a result, while doing my internship emergency rotation, every time a patient presents with an eye issue, I would secretly pray: “Please not me, not me…” So below are some general dos and don’ts when approaching the eye:
·      History and examination should include these aspects (PLOAFF):
o   Pupillary reactions
o   Lids and ocular adnexa
o   Ocular movement
o   Acuity
o   Fundus
o   Fields to confrontation
·      Never put pressure on the eye!
·      Never use atropine drops to dilate the pupil! Use short-acting mydriatic ONLY if necessary—and never when the ocular state/optic nerve function needs to be monitored.
·      X-ray the orbits when suspecting intraocular foreign body—request X-rays with eyes in up and down gazes.
·      Never use steroid drops in the emergency setting.
·      Do not apply ointment in cases suspected of having a penetrating injury.
·      Do not persist in removing a foreign body is it’s not easily removed.
·      Always provide systemic analgesia in cases of corneal injury.
·      Always pad an eye after instilling local anesthetic.
·      Always refer children with squints!

Regarding the use of a slit lamp, I’m kind of ashamed to say that I’ve only ever used it to check for corneal ulceration. I don’t really know all the other uses of the slit lamp and how to perform these tests; so I’ve looked up some useful videos which go through all the basics of the slit lamp use. (The third one is a gem.)

Moving on to some of the emergency presentations involving the eyes. These can be divided into approximately five big categories: foreign body, burns, trauma, painful red eye, and sudden loss of acuity.

Obviously, if there are foreign bodies within the eye, the goal is to locate and remove that object. Foreign bodies can be present in the lid, conjunctiva, cornea, and intraocular (remember to get that orbital X-ray!); they often cause abrasions too so check thoroughly with fluorescein. The main management is to rinse with lots and lots of saline, removal of foreign object, antibiotic drops qid, and tetanus prophylaxis.

The management of chemical burns is very similar to managing skin burns—first rinse and rinse with lots of saline! Local anesthetic and systemic analgesia can be used. Examine thoroughly to make sure all source of the chemical is removed. In thermal burns the principles are similar; start antibiotic drops and pad if possible. Flash burns can result from UV light, and the pain can start up to 6~12 hours after the injury and last for 24 hours; systemic analgesia, sedation, antibiotic ointment, and padding are usually needed.

Blunt force trauma can happen anywhere on the eye, and the history is not a good guide to the severity of the injuries—so beware! The key is to determine where the bleeding is! Although the subconjunctival hemorrhage looks really bad, it’s actually not that bad; only reassurance is needed. The hyphaema doesn’t look that bad in contrast, but it can cause secondary glaucoma; and there can be a more severe secondary hemorrhage 2~3 days later. This requires urgent ophthalmology consultation within 24 hours. Remember to avoid aspirin. Vitreous hemorrhage and choroidal hemorrhage also need consultation within 24 hours to exclude retinal detachment. The most urgent of all eye hemorrhages is the orbital hemorrhage, which usually is accompanied by blowout orbital fracture (RESTRICTION OF MOVEMENT AND DOUBLE VISION)—this is sight threatening and may need urgent decompression—especially if there is reduced vision, nonreactive pupils, or proptosis.

The painful red eye is a very scary topic. There are two ways of approaching it. The first way is to divide it into two big categories: inflammation or glaucoma. Inflammation would include acute conjunctivitis, acute keratitis (HERPES SIMPLEX ULCERATION), acute iritis (CILIARY INJECTION), and orbital cellulitis. Essentially the management is to swab the eye and start antibiotics/antiviral therapy; dilate the eye and give steroid drops for acute iritis. Acute narrow-angle glaucoma is sight threatening, so must be considered in all unilateral painful red eye. Tell-tale symptoms and signs are HALO AROUND LIGHTS, FIXED MID-DILATED PUPILS, nausea and vomiting, SHALLOW ANTERIOR CHAMBER, and increased intraocular pressure (>40mmHg). Treat with topical beta-blockers, adrenergics, and cholinergics and systemic carbonic anhydrase inhibitors and hyperosmotic agents. Refer urgently.

The second way to approach the red eye is by categorization of accompanying symptoms:
·      Light sensitivity—iritis, keratitis, abrasion, ulcer
·      Unilateral—as above + herpes simplex, acute angle closure glaucoma
·      Significant pain—as above + scleritis
·      White spot on cornea—corneal ulcer
·      Blurred vision—all of the above
·      Non-reactive pupil—acute glaucoma, iritis
·      Copious discharge—gonococcal conjunctivitis


Sudden loss of vision in a “white eye” usually means an occlusion of a blood vessel or retinal detachment. Retinal artery occlusion is usually painless, causing partial/total loss of vision, pale disc, retinal edema, CHERRY RED SPOT, and narrowed arteries; remember to investigate for the CAUSE such as temporal arteritis or emboli. Management includes lowering intraocular pressure (IV acetazolamide 500mg), dilate blood vessels by giving carbagen (95%O2 5%CO2) or rebreathing into paper bag, ESR levels, and urgent referral. Retinal vein occlusion can present also present as painless loss of vision, but there is usually dilated retinal veins with multiple hemorrhages through out retina and swollen disc. HISTORY OF FLASHES AND FLOATERS and partial field loss indicate retinal detachment; the retina is grey, elevated in a “veil-like” manner. Urgent referral needed. Another case to refer urgently is optic neuritis; the loss of vision is variable, but key symptom is CENTRAL FIELD LOSS, afferent pupillary defect, and marked loss of red saturation.

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